This content weaponizes scientific jargon to dress up anecdotal survivorship bias as a biological breakthrough. It fundamentally misapplies Wolff’s Law, conflating bone density remodeling with the biological impossibility of resurrecting fused growth plates.
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Deep Dive
I DISCOVERED HOW TO REOPEN BONE PLATES AND HERE’S HOW IT WORKSAdded:
All right, guys.
I've made history.
Today I'm going to explain [music] how you can reactivate your [ __ ] bone plates, right? This is not something that you can do, um, you know, like everywhere.
But at least I don't know. I haven't tried it in other places. [music] But I know for certain that you can do it in your intervertebral disc and like you know, your freaking vertebral bodies.
And that's because endochondral ossification, which is the process by which your bone plates turn, um, you know, cartilage into actual bone is a bit different [music] in your vertebral column.
Why, you may ask?
Here's the [music] bombshell.
Your bone plates don't close at 15, at 16, and not even at 18.
They usually begin to close at 20, >> [music] >> all right? So think of it in this way.
At zip, all right?
Your bone plates in your vertebral, uh, freaking spine, you know, the cervical spine and in the lumbar spine, they are closing.
All right? So it's going to happen firstly on the extremities and then later on on the thorax, all right? So, around 20 to 22 the bone plates in, uh, your lower bodies are going to close. For some people it can happen a little bit later. So, for example, if for you happen to have a late growth spurt where your body hasn't gone through the full extent of what it should be relative to your age, you don't have beard, you maybe still have a baby voice or whatever, so you haven't fully matured yet or you perhaps grew like an inch or half an inch in, um, in your 20s, whatever, there's a really strong likelihood that these bone plates are might actually stay open for a little bit longer then for you, all right?
So, that's the first thing.
And the reason why I'm saying that this is super exciting is because they might actually stay open until you are 25 or perhaps even 27, >> [music] >> right?
And that I think is definitely my case.
The reason why I think I've managed to grow taller is because my bone plates were still open.
But, what's even more interesting is [music] that through intervertebral manipulation, I managed to reactivate it on plate.
Right? Because [music] I think that a lot of people have these bone plates still open. Like I said, the thorax.
So, yeah, initially in this whole compartment here, your bone plates close, [music] you know, in between 25 to maybe even, you know, right before the third decade for some people. They conducted a study in Portugal and they saw that 5% of people had them open until 27. So, you know, it is quite significant. [music] So, them being open means that we can work upon the walls. So, we can induce end organs and we can modulate the expression of the growth [music] plates through the Utah Welcome principle.
Right? So, when you are alternating in between the cyclic compression and decompression sheet, you are pretty much creating an environment where the intervertebral discs, which are avascular, by the way, which means that they aren't actually able to carry nutrients in the same way that all the tissues are. So, for example, your bones, even though they are really hard and uh hard to like, you know, reshape, they actually uh have the ability to carry nutrients, you know, they can absorb calcium, phosphorus, and, you know, all the other minerals that it needs in a much better and fast way.
But, unfortunately, the cartilage can't because, you know, especially the intervertebral disc, there's literally nothing else that the cartilage can interact with other than the end plate, which is then connected to the bone marrow of your vertebral bodies, right? So, that's that's the only gateway, but it's literally a halfway step, right?
And that's why I'm saying that it is super cool because now instead of only applying the diffusion, which is the process by which you know, solutes move from high concentration to low concentration, which is exactly the process by which you know, you lose height at the day, you know, as your body simply lose all of that [ __ ] that it's trying to retain, right?
You are actually now able to engage in active convection, right? Which is the principle by which the body is now not just, you know, exchanging solutes, but actually, you know, exchanging nutrients and even liquids, right? So, this is going to go in and out. Whenever it goes in and it stays in, nutrients are being refined. So, you can think of it like, you know, as the metabolism of the intervertebral discs, right?
And as it metabolizes things, you produces waste, right?
And the waste obviously prevents new nutrients to flow in again and work at the same efficiency, right?
So, you have to think of it like as a filter. You know, whenever you are going through the decompression and you know, inducing in the um nutrient uptake within the intervertebral disc, you are depositing waste, right? Think of it like as a river that has a lot of freaking rocks, you know, that is now blocking the flow, right?
So, that's why we have compression.
Compression forces the discs to squeeze out everything, you know? Some of the byproduct, obviously, some of the nutrients that [music] it hasn't fully refined yet, but it makes possible for a new cycle to begin again, which can then induce in uh uh more proteoglycan synthesis, right? And a chondrocyte differentiation.
Which means that since the bone plates are open you can make [music] your growth plates work again.
And that's the [ __ ] difference.
Your bone plates aren't closed. They are just inactive. So, if you're in between 20 to 25, >> [music] >> there is a really strong likelihood that you could reactivate your bone plates, which means that you're also going to upregulate osteoblast recruitment in the process and really supercharge your intradiscal [music] behavior because, you know, more chondrocytes that are produced not just by the annulus pulposus, right, but also by the end plate means that there is going to be a much bigger degree of um you know um adaptation, right?
Despite the avascularity of your intervertebral body, which is super insane, right? And also on top of this, we also can act upon our Wolff's law because, you know, we talk about the the decompression and compression cycle, but the coolest thing about this [ __ ] is that whenever this cycle occurs, right? So, whenever we are compressing through, you know, the pogo jumps through, like, you know, the squats, the Bulgarian splits, or any of the other exercises, >> [music] >> we are to a small extent inducing, you know, whole body shock wave, okay? And when you go in inducing a shock wave, you're basically inducing a squeeze of the bone marrow.
And when the bone marrow is squeezed, [music] we activate Wolff's law, which then inducing a osteoblast recruitment, all right? So, now this new cartilaginous tissue that we have formed in the epiphyseal plate can turn into actual bone through, you know, this endochondral ossification. So, we are effectively re-fucking opening the bone plates even though we aren't. So, like, now we are activating them. So, they went from being off because, yes, there is no enough chondrocyte proliferation, there is pretty much no osteoblast recruitment to actually having both of them active, and then we are modulating all of these things through the Wolff-Tel Volkmann principle because, now, instead of just, you know, doing nothing, we're actually actively skewing the body not towards cortical thickening, which can happen, obviously, to these exchange of nutrients, right? When you're doing compression work, you are actually inducing cortical thickening alongside density changes.
But, here since we are biasing the spine towards lengthening, instead of the cortical thickening occurring, we are actually gaining longitudinal growth.
So, this is exactly what I discovered.
And the reason why I'm so ecstatic, and I think this is a pretty much like, you know, beginning of a new paradigm, is because if you can induce these in the intervertebral discs and in general in the vertebral bodies, then >> [music] >> we can 100% apply the same principle to a smaller extent to all the you know all the areas that >> [music] >> that have the bone plates, right? So, for example, if your bone plates in your femurs or your tibia are still not fully fused, then there is a good chance that perhaps by inducing these cyclic compression and decompression, you might be able to supercharge a little bit your intradiscal behavior, right?
And it's actually so much easier when you [music] think of it like in in other extremities. You see, I'm formulating this as I'm speaking, by the way. But, like I started thinking about, you know, things like blood flow restriction, for example, or even the clamping [ __ ] you know, LSJL. And those are all things that are pretty [music] much supercharging the the bone plates, right? They are working up on the Uth and Volkmann principle.
So, if you are doing those things, you know, alongside the cyclic compression and decompression thingy, you might be able to, you know, get so much more out of this whole process while your bone plates are open.
And even if they think if you think that they are closed, they might still be in the you know um hibernating phase.
>> [music] >> And therefore, you might still be able to induce in enough endochondral um ossification in the process through, you know, the lengthening [ __ ] and inducing enough chondrocyte recruitment to know you aren't going to actually rejuvenate your bones or whatever, but you are going to be able to get the absolute most out of the process and by extension >> [clears throat] >> be able [music] to reawaken them plates, right? I don't know if that makes any sense, but obviously there are some timelines by which is this possible or not. Like if you think that your growth plates are closed and if you haven't grown in like three or four years, then it's [ __ ] wraps, don't worry. But if it's been like a year or two since you stopped growing like you know it was in my case like I stopped growing at 20 and uh yeah, right before I turned 23, I started you know trying to look more into this [ __ ] So yeah, that was like two years and some change. So yeah, maybe I'm not saying that you should use myself as an indicator because you know it's it can be [ __ ] super different person person and there is no real clinical science or data yet to like you know >> [music] >> actually uh help anybody to make any freaking educated conclusions out of like you know the hypotheticals. Nonetheless, you know you can use me as patient zero. So if you haven't grown in like you know two years or perhaps two and a half, you might be able to still be able to do it through this thingy. So you can try and and see for yourself, right?
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